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Glyceryl trinitrate is extremely effective at relieving the pain of angina but it has only a short half-life. Due to extensive first pass metabolism the drug is ineffective when taken orally and is administered sublingually or buccally in either tablet or aerosol form. Sustained release forms are available to provide prophylactic action against angina attacks; these are formulated into transdermal patches.

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Sor. orgmemberseffective-practicepres rad newsnews A physiotherapy perspective of non-medical prescribing is discussed here. Like podiatrists and radiographers, physiotherapists can, under PSDs, currently supply andor administer medicines to a named patient and under PGDs they can currently supply andor administer medicines to groups of patients who may not be individually identified prior to treatment. Many physiotherapists are already using PSDs and PGDs as named individuals within their current practice. There may be many more that are not, but perhaps should be doing so, because often physiotherapy intervention can be improved by timely administration of medicines by a therapist and outcomes of long-term care can also be enhanced. The use of PSDs in physiotherapy practice is illustrated by situations such as the post-operative administration of oxygen by a physiotherapist on the direction of an anaesthetist or the administration of or advice on the use of, analgesia prescribed by a doctor to a patient with rheumatoid arthritis.

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Methylprednisolone has recently been added to the list of drugs that registered podiatrists can administer by injection into soft tissue injury. Registered physiotherapists can administer corticosteroids by injection according to patient group directions. use of patient group directions by health care professionals. Disadvantages of systemic corticosteroids are widely recognized and perhaps the most serious is suppression of the pituitary-adrenal axis. The addition of exogenous corticosteroids to the systemic circulation is detected by the pituitary gland. In response the pituitary gland reduces its own release of adrenocorticotrophic hormone.

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